RESTORE Study Design

Plasma cholestanol and urine 23S-pentol were assessed at multiple time points over 24 weeks of treatment.1

Significant difference between CTEXLI and placebo seen within 4 weeks1

4x

lower level of plasma cholestanol with continued CTEXLI treatment vs placebo (change from baseline to Day 29)1

(CTEXLI –2.3 μ/mL vs placebo 6.2 μ/mL; estimated treatment difference: –8.5 μ/mL [95% CI: –13.2, –3.9])

Cholestanol levels over 24 weeks graph

For each study treatment (placebo or CTEXLI), the mean value at baseline was calculated as the mean of the measurements obtained prior to receiving the study treatment during the double-blind study duration. The mean value at Day 29 was calculated as the mean of the measurements at Day 29 at the end of the study treatment.1

For each patient at each visit, the measurement of urine 23S-pentol was calculated as the geometric mean of the first 3 morning void urine samples collected within 5 days prior to the visit.1

Significant reductions during the 8-week, open-label CTEXLI phase (P=0.0012)3,4

150x

lower level of urine 23S-pentol with continued CTEXLI treatment vs placebo (change from baseline to Day 29)1

(CTEXLI 185 ng/mL vs placebo 29,506 ng/mL; estimated treatment difference: –29,321 ng/mL [95% CI: –45,701, –12,941])

Within 8 weeks of starting the open-label CTEXLI phase, significant reductions in urine 23S-pentol were seen in both groups (P=0.0012).1

23S-pentol measurements during the 8 week open-label CTEXLI phase
Adherence is critical

Adherence is critical: significant increases in toxic bile acid precursors were seen within 2 weeks of stopping CTEXLI4

These significant increases were seen when patients switched from CTEXLI to placebo in the double-blind withdrawal periods.